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Computational Identification of Alzheimer's Disease Specific Transcription Factors using Microarray Gene Expression Data

Vishalini Krishnamurthy, Nicy Sweety Issac and Jeyakumar Natarajan*

Data and Text Mining Laboratory, Department of Bioinformatics, Bharathiar University, Coimbatore 641046, India
*Corresponding author: Dr. N. Jeyakumar, Data and Text Mining Laboratory,
Department of Bioinformatics, Bharathiar University,
Coimbatore 641046, India,
E-mail: n.jeyakumar@yahoo.co.in
Received November 05, 2009; Accepted December 18, 2009; Published December 20, 2009
Citation: Krishnamurthy V, Issac NS, Natarajan J (2009) Computational Identification of Alzheimer’s Disease Specific Transcription Factors using Microarray Gene Expression Data. J Proteomics Bioinform 2: 505- 508. doi:10.4172/jpb.1000113
 
Copyright: ©2009 Krishnamurthy V, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
 
Abstract
Alzheimer’s disease is the most common form of dementia affecting millions of older people world wide. Identification of transcriptional factor binding sites of disease specific co-expressed genes and the possible transcriptional regulation of the genes will lead to a better understanding of complex diseases such as Alzheimer’s disease. However, the regulatory mechanisms driving these changes, in particular the networks of transcription factors involved, is not fully understood to date. The computational identification of conserved TFBS in the regulatory regions of hundreds of genes at a time especially suited for microarray gene expression datasets. We report clusters of co-expressed genes and the identification of conserved TFBSs using microarray gene expression data sets. We investigated microarray gene expression data from Gene Expression Omnibus (GEO) specific to Alzheimer’s disease. The dataset consists of 14 normal and 14 Alzheimer disease samples. Differential expression analysis results 240 differentially expressed genes which are more significant. Hierarchical clustering of these significance genes shows eight clusters of co-expressed genes. The detection of over-represented transcription factor binding sites in the promoters regions of co- expressed genes reveals transcription factor binding site classes ZEB1, MZF1 1-4, ZNF354C, ELF5 and SPIB in upstream of human promoter and responsible for apoptosis.
 
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